a) There is a history of trauma to the ankle
followed by synovial swelling of the ankle joint, infilling the posterior
aspect of the joint bilaterally and allowing ballottement of the fluid.
b) Passive ankle flexion, extension and talar
translation hurt in a capsular pattern, but trauma to the ankle usually
disturbs the ligaments and tendons as well so that, clinically, there will be
accompanying signs from these to confuse the diagnosis.
c) Inflammatory disease has no history of trauma,
therefore no individual ligamentous signs are found, but there is likely to be
a capsular pattern of pain accompanied by systemic stigmata.
Cause
Sprain of the talar
joint capsule and its ligamentous thickenings. Inflammatory joint disease.
Treatment
b) Electrotherapeutic modalities to settle
inflammation, such as interferential and pulsed shortwave diathermy
c) Non-steroidal anti-inflammatory drugs
d) If systemic causes are suspected, then
diagnostic aspiration of the joint, which must be sent for culture and crystal
microscopy.
e) Injection of cortisone to settle capsular
inflammation, via either the anterior or posterior approach.
f) Isometrics to the posterior tibialis and
peroneals, to maintain strength.
g) Balancing or wobble board exercises, for
proprioceptive skills.
h) Non-impact cross-training, such as swimming,
cycling and rowing routines.
Sports
Following injury, most
ankles will require supporting for 4-6 weeks after reaching match fitness.
If this is a traumatic
inflammatory lesion then early cortisone injection settles it and permits
earlier rehabilitation. However, if possible, take an aspirate before the
injection, for culture and polarized light microscopy. Though a bone scan can be
the watershed investigation separating bone and soft tissue injuries, MRI and
especially MRI arthogram, is the most definitive investigation for the majority
of ankle lesions.
"Concise
guide to sports injuries, 2nd edition",Churchill Livingstone,
Malcolm T.F. Read, foreword by Bryan English
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